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Peyronie's Disease: Peyronie's Disease (pay-row-KNEES) is acquired in adult life rather than at birth (like hypospadias). Men with Peyronie's disease usually seek medical attention because of painful erections and difficulty with intercourse. Peyronie's Disease is caused by the formation of hard plaque on the upper or lower side of the penis. The plaque forms in layers containing erectile tissue. The local inflammation eventually develops into a full scar. This scar tissue is hard and inflexible, and causes the penis to bend when erect. The effects of this bend are far less noticeable when the penis is in a flaccid state. François de la Peyronie, a French surgeon first described Peyronie's disease in 1743. Early writers classified it as a form of impotence, now called erectile dysfunction (ED). Peyronie's disease can be associated with ED; however, experts now recognize ED as only one factor associated with the disease, a factor that is not always present. This affliction leaves the penis bent distinctly in some direction. Often, the angle is in excess of 45 degrees and results in serious pain during erection and the impossibility of normal sexual intercourse. Bear in mind that a gentle curve in the penis is natural and you probably don’t have Peyronie's Disease! There is no need to worry about a curved penis unless the curve suddenly appeared and/or you're penis has experienced some sort of trauma. Unfortunately, symptoms of the disease may develop methodically or overnight. Overnight appearances are usually due to some sort of serious penile trauma, but not always. If you think that you have Peyronie's Disease, then I highly recommend you see your healthcare provider, who may refer you to an urologist. Hypospadias: Hypospadias is a congenital disorder of the penis, an abnormality that may affect up to one out of every 400 to 500 male infants. Instead of having a urethral opening at the end of the penis, boys born with hypospadias have an opening on the underside of the shaft of their penis. If this causes a problem with urination, the condition can be surgically corrected. About 10% of boys born with this defect may also have undescended testicles. Surgery is most often performed before the child reaches school age. The surgery involves creating a tube to extend the urethra to the end of the penis. The original hole is most often left as it is since the urethra now bypasses it. Although the penis has two holes, only one is functional. Otherwise it should be a normal functioning penis. Priapism: Priapism is an involuntary prolonged or painful erection that can persist for hours, days and is not associated with sexual arousal. It can occur at any age and is a true emergency with risks of subsequent impotency. Primary priapism is the result of trauma or infection. Secondary causes include sickle cell disease, spinal cord injury and stroke. Various medications can also contribute to this condition. Phimosis: Phimosis refers to a tightening of the skin of the foreskin that prevents retraction over the glans—the sensitive erectile tip of the penis. There are two typical forms of this tightness: an infant phimosis and the phimotic ring or band. An infant phimosis has an easily recognizable tubular form, this is common and healthy in infants but occasionally will continue into adulthood. The adult phimosis is a thin contour of skin tissue located towards the front of the inner foreskin and it narrows the opening of the foreskin. A phimotic ring can make retraction of the foreskin over and behind the glans impossible, painful, or difficult, the foreskin may even get stuck behind the glans. This condition is often treated by circumcision, however, there are less invasive procedures depending upon the degree of phimosis, see several urologists for opinions if you have this condition. For “simple” phimosis, stretching of the foreskin may be a method for treatment that may work. Steroids and surgery are other options. For more on this condition do a search on google. Large Penis Veins: It’s normal for men to have prominent veins on their penis. For some men, sometimes the appearance of veins is a result of poorly functioning valves in their testicles. If you see your veins changing in size or color, it’s time to see your friendly urologist. male penile enlargement free penis elargement technique penis enargement information penile enlargment operation buy penis enlarement pills free penile enlargement exercise penile enlargment surgery magna rx results review

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If you have taught your child all the rules of ‘stranger danger’ you have protected him/her from a 1% chance of being sexually abused. This leaves your child vulnerable to the most likely sexual child abuse offender, family members or other trusted adults. 80% of children are sexually abused by a family member, 19% are abused by someone the child knows and trusts. The other little known statistic is the frequency of sexual child abuse. David Finkelhor and Dianna Russell’s research reveals 62% of girls and 31% of boys will be sexually abused by age 18. Unfortunately this statistic is considered low due to the difficulty in gathering data through surveys or reporting agencies. For many decades we have screamed, ranted, condemned, demanded and enacted legislation to punish sex offenders to little avail. The news media and magazines have joined in the campaign to illuminate the problem after the damage is done. As a result of the media’s incessant coverage and hype of ‘strangers,’ we have come to believe if we teach our children about ‘stranger danger,’ we have thoroughly protected our children from this horrific crime. The first response we form when hearing of sexual abuse or incest is denial. ‘I don’t have to be concerned about that in my community. That would never happen in my family.’ The unbelievable reality is that a person who sexually abuses children may seem very average and ordinary to the world. Furthermore, we find sexual abuse and incest even more difficult to believe or accept when the person we like, admire, love, and/or marry is the perpetrator of the abuse. Tragically, the unwillingness to accept the facts concerning sexual abuse perpetrators leaves children vulnerable to becoming victims and increases the likelihood that they will be abused. To understand how sexual child abuse is perpetrated by the person we least suspect one needs to have a comprehensive definition of sexual abuse. “Traditionally, incest [sexual abuse] was defined as: sexual intercourse between two persons too closely related to marry legally--sex between siblings, first cousins, the seduction by fathers of their daughters. This dysfunctional blood relationship, however, does not completely describe what children are experiencing. To fully understand all sexual abuse, we need to look beyond the blood bond and include the emotional bond between the victim and his or her perpetrator. Thus, a new definition has emerged. The new definition now relies less on the blood bond between the victim and the perpetrator and more on the experience of the child. Incest is both sexual abuse and an abuse of power. It is violence that does not require force. Another is using the victim, treating them in a way that they do not want or in a way that is not appropriate by a person with whom a different relationship is required. It is abuse because it does not take into consideration the needs or wishes of the child; rather, it meets the needs of the other person at the child’s expense. If the experience has sexual meaning for another person, in lieu of a nurturing purpose for the benefit of the child, it is abuse. If it is unwanted or inappropriate for her age or the relationship, it is abuse. Incest [sexual abuse] can occur through words, sounds, or even exposure of the child to sights or acts that are sexual but do not involve her. If she is forced to see what she does not want to see, for instance, by an exhibitionist, it is abuse. If a child is forced into an experience that is sexual in content or overtone that is abuse. As long as the child is induced into sexual activity with someone who is in a position of greater power, whether that power is derived through the perpetrator’s age, size, status, or relationship, the act is abusive. A child who cannot refuse, or who believes she or he cannot refuse, is a child who has been violated.. (E. Sue Blume, Secret Survivors).” There are two types of sexual abuse approaches—overt and covert. Overt sexual abuse is openly sexual and apparent. Although there may be an attempt to deny that it is abusive, there is no attempt to hide the fact that it is sexual in nature. Covert sexual abuse is more insidious. Thus, identifying it is harder, because the sexual nature of the action is disguised. The perpetrator acts as if she/he is doing something non-sexual, when in fact he or she is being sexual. The betrayal then becomes two-fold. The child is not only abused, but also tricked or deceived about the act. In this dishonesty, the child is unable to identify or clarify his/her perception of the experience. The unreal or surreal sense that accompanies any sexual abuse is intensified when the child is tricked into disbelief. Thus, the child doubts his/her perceptions and feelings and believes that there is something wrong with him/herself because he/she feels terrible. To make matters worse, everyone around her/him discounts signs of the abuse, because we don’t want to believe someone with a sterling public image would do such a thing. Thus the child feels crazy, as if she/he is the one with the problem. One example of overt sexual abuse whereby the perpetrator disguises his actions and those present are in denial about what is transpiring is exemplified by the incident a client, who is a sexual abuse survivor, reported seeing. Her father (her perpetrator) kissed his granddaughter, her one-year-old niece on the pubic area after her niece finished her bath. Her sister, the child’s mother, the child’s grandmother (wife of the perpetrator) were present. “My sister and mother (the child’s grandmother) laughed and I got sick to the stomach. Am I over reacting,” she asked. Obviously, her sister and mother are unaware of the definition of sexual abuse. Except for the fact this woman was in therapy she would not have considered it sexual abuse either. An example of covert sexual abuse by someone we least expect is exemplified by a 39 year-old woman who came to me after having a severe panic attack. During our investigation as to the root cause of the panic attack she revealed she had been ‘fondled’ when she was nine by a family friend. “He helped me on with my coat at a family gathering. As he adjusted my coat onto my shoulder, he fondled my breast.” This type fondling is often times referred to as ‘coping a feel.’ No matter the label, it is sexual abuse and causes damage. Women know how icky it feels when a man ‘cops a feel.’ Can you imagine what it would feel like for a nine-year-old, who has no information to comprehend and emotionally resolve what she experienced? Another example of covert sexual abuse by someone you least expect was told to me by my client, Rickie (not his real name). He remembered being held by his mother’s best friend in the water at the beach when he was six, while his parents sat on the beach. Fully protected from view by the water, she fondled his penis. This was not the end of the sexual abuse. When Rickie was 15 years old, she enticed him to have sex with her at her home while he waited for her son, his friend to come home. The second incident of her sexual abuse of Rickie was overt. There are six key techniques to abuse-proof your child. •Avoid spanking your child—spanking is a body boundary violation. Perpetrators target children who have had body boundary violations because they are less apt to protest any unacceptable body boundary violations, are more compliant with adults and are less apt to tell. You can avoid your child from falling prey to these cunning perpetrators by doing everything to avoid making your child a target. •Avoid touching your child in erotic areas—buttocks, chest, thighs, etc. Perpetrators state they use familiar touch (rubbing the child’s legs, buttocks or hugging/kissing) to desensitize the child before using touch which is sexual in content and intent. If your child is unaccustomed to being touched in erotic areas, he/she will protest immediately. Protesting will either thwart the perpetrator or alert anyone nearby that something is awry. •Teach your child self-protection by teaching him/her to protest violation of body boundaries or unwanted touch beginning at age two. •Practice and teach your child good body image. •Practice and teach your child to TELL YOU EVERYTHING, NO SECRETS FROM MOMMY and DADDY. •Practice and Teach Appropriate Suspicion—Trust your intuition, (a.k.a Sixth Sense) herbal pnis enlargement pills vimax penis enlargement pills review penile enlargement technique easy enlagement free penis surgery way buy pennis enlargement pills vigrx hoax pro acne solution enlagement erection penis pill vimax free penis enargement tip

The most commonly used drugs that are available to treat balding are Minoxidil and Finasteride. Unlike Minoxidil, which is a topical product, Finasteride is an orally taken prescription drug. It Is to be taken once daily. Both medications have been studied extensively. They’re most successful at promoting re-growth at the vertex of the scalp. Unfortunately, most patients experience little or no results to the hairline or anterior scalp. Ask your family physician if either of these products are right for you. It’s important that you’re descriptive when referring to current medications you’re taking and any allergies or medical complications you may have. It’s quite evident that hair plays an important role in a person’s self image. After all, men are constantly bombarded with ads that portray male sexiness. These ads employ young muscular male models with full heads of hair. But men are not alone! Women too, experience these internal struggles. Much like a woman is ‘supposed to’ look stick thin and youthful, a man is ‘supposed to’ look a certain way: strong and virile. Those who want to play up to the image that they’re ‘supposed to’ look like–can turn the page of their magazine and find another advertisement. Ah, the solution–medication. Nowadays there are medications for everything! Weight loss, breast enhancement, skin pigmentation, penis enlargement, and yes–hair loss and balding. Do these medications actually work? Advertisements will promise you the moon AND a money back guarantee. They may even tell you that you have nothing to lose! Unfortunately, that’s the farthest thing from the truth. Many of these medications rely on steroids, which can promote acne, irritability, insomnia, skin rashes, and…balding and hair loss. Imagine that? A hair loss product which has the side effect of balding. It happens! In addition, a product which declares its ingredients “all natural” hardly ever lists the exact contents and their measurements on the bottle’s label. Products ‘like’ or ‘similar to’ Minoxidil and Finasteride are usually derived from companies that like to ride on the coat tails of others. If you’re willing to give it a shot, do your homework and research the company and find testimonials from other men like yourself. Why waste your money and time trying product after product? Never purchase a product on a whim. Blind purchases often leave bald men victimized. Contact the owner of the site who manufactures the product and ask directly. Inhairit offers an all natural solution to hair loss with minimal side effects. penis enlarement information penis enhancement medical penis enlagement penis enlagement review truth about penis enhancement pills penis enlargment program medical penile enlargement herbal natural penile enlargement free penis enargement tip

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enlargment procedure homemade penis enhancement free penis enargement penis enlagement pills review vigrx oil vimax penis enlargement cream pennis enlargement herb penis enlagement supplement free penis enargement tip

Pumpkin is at the heart of Halloween festivities, and for many is the only experience they have of the squash all season. This is usually for the joys of pumpkin carving rather than cooking. When you discover just how healthy pumpkins are however, you'll realise that pumpkin recipes are just as worth getting excited about and that it would be foolish not to eat the remaining flesh left over from your pumpkin carving. (As a note, if you're rolling up your sleeves ready to tackle your orange fellow there are plenty of free pumpkin carving pattern ideas available on the internet). Pumpkin is a superfood indeed. In fact, it's not just the inner meat of a pumpkin that's virtuous but its seeds are super nutritous too and contain a wealth of health-promoting properties. Like melons, cucumber, and squash, pumpkins belong to the gourd family. They were much celebrated by Native American Indians who made use of their flesh and seeds for culinary and medicinal purposes. Pumpkin flesh is high in fibre and vitamins C and E, magnesium and potassium and a staggering quantity and variety of carotenoids, being one of the most abundant natural sources of these amazing phytonutrients. Dietary carotenoids correlate with lower a risk of numerous cancers, heart disease, cataracts and blindness. Pumpkin contains heaps of beta-carotene as well as a huge concentration of alpha-carotene, with just a quarter of a cup yielding the recommended daily amount. Alpha-carotene protects against certain cancers and cataracts, and has also been discovered to be a powerful ally against aging. Both alpha- and beta-carotene can be converted into vitamin A within the body, which plays an essential role in growth, development and immunity. Beta-carotene is a potent antioxidant and anti-inflammatory agent and prevents cholesterol build up in blood vessels that could lead to stroke or heart attack. The presence of magnesium, potassium and folate in pumpkin heightens its heart-friendly attributes. Beta-carotene works most effectively in combination with other carotenoids and has been found to reduce the risk of lung and colon cancer in particular. When exploring pumpkin recipes experiment with other squashes too. There are numerous types in all shapes and sizes with many similar nutritional values. One tasty alternative is the butternut squash. Pumpkins yield delicious and highly nutritious seeds. Known as pepitas, they are dark green and flat with a chewy texture and rich nutty flavour. These seeds are dense in minerals, with just a quarter of a cup providing approximately half the daily recommended dose for magnesium and iron, in addition to high doses of zinc, phosphorous, potassium, selenium, manganese and copper. They also contain the amino acid tryptophan known for anti-depressant qualities, and essential fatty acids. The dark green oil produced from pumpkin seeds has been used throughout history in India, Europe and America to fight parasites, aid the digestive tract and help with prostate and reproductive disorders. It has also been recommended for pregnant and lactating women because of its high content of essential fatty acids. Pumpkin seeds have been found to help prevent against prostate gland enlargement due to the chemical substances called cucurbitacins it contains. The essential fatty acids in pumpkin seeds are also necessary for prostate health, and zinc (which pumpkin seeds are especially high in) is great for the reproductive systems and has been shown to reduce prostate size. The L-tryptophan in pumpkin seeds is known to have anti-depressant properties, so it is suggested eating them can elevate your mood. Pumpkin seeds also contain omega-3 fatty acids known for their role in improving mental function, alleviating depression and aiding memory. Both the omega-3 and omega-6 essential fatty acids in pumpkin seeds have a broad range of health functions in the body. So, for a smashing pumpkin Halloween, as well enjoying carving pumpkins, check out some tasty pumpkin recipes so you can benefit from the super healthy flesh of your pumpkin carving remains, and throw a handful of pumpkin seeds on top for that extra nutritional boost!